Academic Journal
Q1Acta Neuropathologica
About Acta Neuropathologica
Acta Neuropathologica is a scholarly journal published by Springer Science and Business Media Deutschland GmbH. SCImago 2025 places it in Q1 with an SJR of 4.191 and an H-index of 241.
Its listed coverage is 1961-2026 and its research categories include Cellular and Molecular Neuroscience (Q1); Neurology (clinical) (Q1); Pathology and Forensic Medicine (Q1). The 2025 dataset reports 130 documents and 3605 citations across the latest three-year reporting window.
About Acta Neuropathologica: A Leading Journal in Neuropathology Research
Acta Neuropathologica is a prestigious, peer-reviewed scientific journal that serves as a premier platform for publishing high-impact research in the field of neuropathology. Recognized globally for its rigorous editorial standards and innovative scientific contributions, the journal is a vital resource for researchers, clinicians, and academics dedicated to understanding the pathology of the nervous system.
A Focus on Excellence in Neuropathology
Founded in 1961, Acta Neuropathologica has built a solid reputation for publishing groundbreaking research articles, reviews, and commentaries on a wide range of topics related to the pathology of the central and peripheral nervous systems. The journal emphasizes studies on neurodegenerative diseases, brain tumors, neuromuscular disorders, and developmental neuropathology, with a special focus on molecular and translational research.
Its primary aim is to bridge the gap between basic neuroscience and clinical practice, offering insights that inform diagnosis, treatment, and prevention strategies for neurological diseases. As the prevalence of disorders like Alzheimer’s disease, Parkinson’s disease, and glioblastoma continues to rise globally, the journal’s role in disseminating cutting-edge research has never been more important.
High Impact and Global Recognition
Acta Neuropathologica is published by Springer Nature and boasts an impressive impact factor, consistently ranking among the top journals in both pathology and neuroscience categories. It attracts submissions from leading researchers around the world and maintains a rigorous peer-review process to ensure scientific accuracy and relevance.
The journal is indexed in major databases including PubMed, Scopus, and Web of Science, making its content highly accessible to a global audience. Its high citation rates and academic influence underscore its role as a critical source of information for the scientific and medical communities.
Diverse Article Types and Timely Topics
The journal publishes a variety of article types including:
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Original Research Articles: Detailed studies offering new insights into neurological disease mechanisms.
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Review Articles: Comprehensive summaries of current knowledge and emerging trends.
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Short Communications: Brief but impactful reports on novel findings.
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Editorials and Commentaries: Expert opinions on recent developments in neuropathology.
Acta Neuropathologica frequently features special issues and thematic series that delve into pressing topics such as neuroinflammation, prion diseases, molecular diagnostics, and personalized medicine in neuropathology.
Author and Reader Engagement
The journal’s editorial board is composed of internationally renowned experts who ensure the publication of only the most relevant and robust studies. Authors benefit from a transparent review process, fast publication timelines, and wide dissemination of their work. Readers enjoy open access options and online-first publications, which keep them updated with the latest advancements in the field.
Journal Metrics
Metrics can change by reporting year. Verify time-sensitive values with the publisher or indexing service.
Aims & Scope
About Acta Neuropathologica: Leading the Way in Neuropathology Research
Acta Neuropathologica is a globally recognized, peer-reviewed scientific journal dedicated to the field of neuropathology. Published by Springer Nature, the journal is renowned for its high-impact research articles, reviews, and short communications that advance understanding of diseases affecting the nervous system. With a longstanding history and a commitment to excellence, Acta Neuropathologica serves as a trusted source for clinicians, researchers, and academics worldwide.
High-Impact Research in Neuroscience and Pathology
As a leading journal in neuropathology, Acta Neuropathologica publishes cutting-edge research that bridges the gap between clinical neurology, molecular biology, and pathology. The journal covers a wide array of topics including, but not limited to:
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Neurodegenerative diseases such as Alzheimer’s disease, Parkinson’s disease, and Huntington’s disease
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Brain tumors and neoplastic conditions
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Inflammatory and infectious conditions of the nervous system
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Neuromuscular disorders
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Genetic and metabolic brain diseases
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Molecular and cellular mechanisms of neurodegeneration
By focusing on both basic and translational research, the journal plays a pivotal role in driving innovation and enhancing the scientific community’s understanding of neurological disorders.
Rigorous Peer Review and Global Recognition
Acta Neuropathologica maintains a rigorous peer-review process to ensure the highest standards of scientific integrity and quality. The editorial board comprises leading experts and scientists in neuropathology, neuroscience, and molecular biology. Each submission is thoroughly reviewed to validate its scientific merit, methodology, and potential impact on the field.
Thanks to its reputation for quality and relevance, Acta Neuropathologica consistently ranks among the top journals in pathology and neuroscience, boasting a high impact factor and strong citation metrics. It is indexed in major scientific databases including PubMed, Scopus, and Web of Science.
Audience and Accessibility
The journal caters to a global audience of researchers, pathologists, neurologists, and medical professionals who seek reliable insights and updates on the latest in neuropathological science. With Springer’s robust digital platform, Acta Neuropathologica ensures wide accessibility and discoverability, offering both print and online formats. Open-access options are also available, allowing authors to maximize the reach and visibility of their work.
Advancing Scientific Knowledge
By publishing novel research, in-depth reviews, and groundbreaking discoveries, Acta Neuropathologica plays an essential role in shaping the future of neurological and pathological science. It not only fosters collaboration among researchers and clinicians but also contributes to the development of diagnostic tools, therapeutic strategies, and a deeper understanding of brain diseases.
Final Thoughts
Whether you are a scientist investigating the molecular mechanisms of neurodegeneration or a clinician seeking the latest diagnostic breakthroughs, Acta Neuropathologica offers a comprehensive and authoritative resource. With its commitment to innovation, integrity, and scientific excellence, the journal continues to be a cornerstone in the world of neuropathology research.
Recent Research Articles
Latest publications matched automatically by ISSN.
Inter-alpha-trypsin inhibitor heavy chain H3 is a potential biomarker for disease activity in myasthenia gravis
Christina B. Schroeter, Christopher Nelke, Frauke Stascheit, Niklas Huntemann et al.
2024-06 · DOI: 10.1007/s00401-024-02754-6Co-registration of MALDI-MSI and histology demonstrates gangliosides co-localize with amyloid beta plaques in Alzheimer’s disease
Nikita Ollen-Bittle, Shervin Pejhan, Stephen H. Pasternak, C. Dirk Keene et al.
2024-06 · DOI: 10.1007/s00401-024-02759-1Annexin A11 aggregation in FTLD–TDP type C and related neurodegenerative disease proteinopathies
John L. Robinson, EunRan Suh, Yan Xu, Howard I. Hurtig et al.
2024-06 · DOI: 10.1007/s00401-024-02753-7Comprehensive assessment of TDP-43 neuropathology data in the National Alzheimer’s Coordinating Center database
Davis C. Woodworth, Katelynn M. Nguyen, Lorena Sordo, Kiana A. Scambray et al.
2024-06 · DOI: 10.1007/s00401-024-02728-8Correction to: Disruption of MAM integrity in mutant FUS oligodendroglial progenitors from hiPSCs
Yingli Zhu, Thibaut Burg, Katrien Neyrinck, Tim Vervliet et al.
2024-06 · DOI: 10.1007/s00401-024-02748-4Associations of CSF BACE1 with amyloid pathology, neurodegeneration, and cognition in Alzheimer’s disease
Feng Gao, Mengguo Zhang, Qiong Wang, Ming Ni et al.
2024-06 · DOI: 10.1007/s00401-024-02750-wBrain vasculature accumulates tau and is spatially related to tau tangle pathology in Alzheimer’s disease
Zachary Hoglund, Nancy Ruiz-Uribe, Eric del Sastre, Benjamin Woost et al.
2024-06 · DOI: 10.1007/s00401-024-02751-9Seeding activity of human superoxide dismutase 1 aggregates in familial and sporadic amyotrophic lateral sclerosis postmortem neural tissues by real-time quaking-induced conversion
Justin K. Mielke, Mikael Klingeborn, Eric P. Schultz, Erin L. Markham et al.
2024-06 · DOI: 10.1007/s00401-024-02752-8Clinically unfavorable transcriptome subtypes of non-WNT/non-SHH medulloblastomas are associated with a predominance in proliferating and progenitor-like cell subpopulations
Konstantin Okonechnikov, Daniel Schrimpf, Jan Koster, Philipp Sievers et al.
2024-06 · DOI: 10.1007/s00401-024-02746-6Ferroptosis inhibitor improves outcome after early and delayed treatment in mild spinal cord injury
Fari Ryan, Christian Blex, The Dung Ngo, Marcel A. Kopp et al.
2024-06 · DOI: 10.1007/s00401-024-02758-2The necroptosis cell death pathway drives neurodegeneration in Alzheimer’s disease
Sriram Balusu, Bart De Strooper
2024-06 · DOI: 10.1007/s00401-024-02747-5Early and selective localization of tau filaments to glutamatergic subcellular domains within the human anterodorsal thalamus
Barbara Sárkány, Csaba Dávid, Tibor Hortobágyi, Péter Gombás et al.
2024-06 · DOI: 10.1007/s00401-024-02749-3Alteration of gene expression and protein solubility of the PI 5-phosphatase SHIP2 are correlated with Alzheimer’s disease pathology progression
Kunie Ando, Fahri Küçükali, Emilie Doeraene, Siranjeevi Nagaraj et al.
2024-06 · DOI: 10.1007/s00401-024-02745-7Xenografted human iPSC-derived neurons with the familial Alzheimer’s disease APPV717I mutation reveal dysregulated transcriptome signatures linked to synaptic function and implicate LINGO2 as a disease signaling mediator
Wenhui Qu, Matti Lam, Julie J. McInvale, Jason A. Mares et al.
2024-06 · DOI: 10.1007/s00401-024-02755-5Alternatively spliced ELAVL3 cryptic exon 4a causes ELAVL3 downregulation in ALS TDP-43 proteinopathy
Isabel Costantino, Alex Meng, John Ravits
2024-06 · DOI: 10.1007/s00401-024-02732-yNeuropathological findings in Down syndrome, Alzheimer’s disease and control patients with and without SARS-COV-2: preliminary findings
Ann-Charlotte E. Granholm, Elisabet Englund, Anah Gilmore, Elizabeth Head et al.
2024-06 · DOI: 10.1007/s00401-024-02743-9Disentangling the heterogeneity of multiple sclerosis through identification of independent neuropathological dimensions
Alyse de Boer, Aletta M. R. van den Bosch, Nienke J. Mekkes, Nina L. Fransen et al.
2024-06 · DOI: 10.1007/s00401-024-02742-wThe influence of APOEε4 on the pTau interactome in sporadic Alzheimer’s disease
Manon Thierry, Jackeline Ponce, Mitchell Martà-Ariza, Manor Askenazi et al.
2024-06 · DOI: 10.1007/s00401-024-02744-8Transmission experiments verify sporadic V2 prion in a patient with E200K mutation
Hitaru Kishida, Atsushi Kobayashi, Kenta Teruya, Hiroshi Doi et al.
2024-06 · DOI: 10.1007/s00401-024-02738-6Identification of high-performing antibodies for the reliable detection of Tau proteoforms by Western blotting and immunohistochemistry
Michael J. Ellis, Christiana Lekka, Katie L. Holden, Hanna Tulmin et al.
2024-06 · DOI: 10.1007/s00401-024-02729-7Reviews
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April 21, 2025 at 5:26 am
April 21, 2025