Academic Journal
Q1Nature Metabolism
About Nature Metabolism
Nature Metabolism is a scholarly journal published by Nature Research. SCImago 2025 places it in Q1 with an SJR of 9.602 and an H-index of 113.
Its listed coverage is 2019-2026 and its research categories include Cell Biology (Q1); Endocrinology, Diabetes and Metabolism (Q1); Internal Medicine (Q1); Physiology (medical) (Q1). The 2025 dataset reports 236 documents and 9553 citations across the latest three-year reporting window.
Nature Metabolism is a high-impact, peer-reviewed scientific journal published by the prestigious Nature Portfolio. Since its launch in 2019, it has rapidly established itself as a key platform for cutting-edge research in the field of metabolism. As the global interest in health, disease, and metabolic disorders continues to grow, Nature Metabolism has become a trusted resource for researchers, clinicians, and healthcare professionals.
What Is Nature Metabolism?
Nature Metabolism focuses on the complex and dynamic processes that govern metabolism at the molecular, cellular, and systemic levels. The journal publishes original research, reviews, perspectives, and commentaries on topics related to metabolic health, including obesity, diabetes, cancer metabolism, cardiovascular health, aging, and the impact of diet and exercise.
The journal stands out for its commitment to scientific excellence, rigorous peer review, and relevance to both basic science and clinical applications. Articles published in Nature Metabolism often present groundbreaking findings that advance our understanding of how metabolism influences overall health and disease progression.
Key Areas of Focus
Nature Metabolism covers a wide range of metabolic processes and pathways. Some of its most prominent research areas include:
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Nutritional metabolism and how dietary factors affect gene expression and metabolic health.
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Mitochondrial function and its role in energy production and cellular health.
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Hormonal regulation of metabolism, including insulin signaling and leptin function.
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Metabolic reprogramming in cancer and immune cells.
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Metabolism and aging, focusing on how metabolic changes contribute to longevity and age-related diseases.
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Microbiome interactions, exploring the link between gut bacteria and metabolic function.
Why Nature Metabolism Matters
In today’s health-conscious world, understanding metabolism is more critical than ever. The rise of metabolic disorders such as obesity and type 2 diabetes has prompted a surge in research and public interest. Nature Metabolism helps bridge the gap between laboratory discoveries and real-world applications, providing insights that can lead to new therapies, diagnostics, and prevention strategies.
For researchers, publishing in Nature Metabolism represents a significant milestone, as the journal is indexed in major databases like PubMed and Scopus and boasts a high impact factor. For healthcare professionals, the journal serves as a reliable source of the latest developments in metabolic research.
SEO Keywords for Target Audience
If you're searching for reliable information on metabolic health, Nature Metabolism should be at the top of your list. Common keywords associated with this journal include:
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Nature Metabolism impact factor
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Nature Portfolio journals
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Journal Metrics
Metrics can change by reporting year. Verify time-sensitive values with the publisher or indexing service.
Aims & Scope
Metabolism is one of the most crucial biological processes that sustain life. It encompasses the chemical reactions occurring within living organisms to maintain life. To fully grasp metabolism, it’s important to explore its scope, nature, and mechanisms. This article will guide you through the fundamental aspects of metabolism, offering insights into how our bodies convert food into energy and maintain cellular function.
Scope of Metabolism
The scope of metabolism covers a wide range of biological activities essential for survival. It includes everything from the digestion of food to the synthesis of DNA. Metabolism is not limited to humans; it is a universal process occurring in all living organisms—plants, animals, fungi, and microorganisms.
In practical terms, metabolism helps in:
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Energy production for growth and cellular maintenance
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Detoxification of harmful substances
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Synthesis of complex molecules like proteins, enzymes, and hormones
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Waste elimination through processes like respiration and excretion
Understanding metabolism also has applications in medicine, nutrition, and fitness. Conditions like diabetes, obesity, and thyroid disorders are directly linked to metabolic dysfunctions. In sports and physical training, metabolic rates are critical for optimizing performance and recovery.
Nature of Metabolism
The nature of metabolism is dynamic and adaptive. It is categorized into two primary types: catabolism and anabolism.
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Catabolism is the breakdown of larger molecules into smaller ones, releasing energy in the process. For example, the digestion of carbohydrates into glucose.
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Anabolism involves building complex molecules from simpler ones, requiring energy. This includes muscle growth and tissue repair.
Metabolism is regulated by various enzymes, hormones, and genetic factors. Enzymes act as catalysts that speed up reactions, while hormones like insulin and adrenaline influence metabolic pathways. Each organism has a unique metabolic profile that depends on age, gender, diet, physical activity, and overall health.
Metabolic Pathways and Energy Conversion
Metabolic reactions occur in a series of steps called metabolic pathways. Some of the most well-known pathways include:
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Glycolysis – the breakdown of glucose to produce energy
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Krebs cycle (Citric Acid Cycle) – a key process in aerobic respiration
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Electron Transport Chain – where most of the cellular ATP (energy) is produced
These processes primarily take place in the mitochondria, known as the powerhouses of the cell. The ATP (adenosine triphosphate) produced is then used for various biological functions, from muscle contractions to nerve signaling.
Recent Research Articles
Latest publications matched automatically by ISSN.
Increasing cytosolic NADPH to mimic exercise
Bryce J. Carpenter, Pieterjan Dierickx
2026-09-08 · DOI: 10.1038/s42255-026-01580-2Pentose phosphate pathway-derived NADPH facilitates physiological hypertrophy and alleviates ischemia–reperfusion injury in the heart
Dan Wu, Tao Chen, Xin Dong, Haoqi Li et al.
2026-09-08 · DOI: 10.1038/s42255-026-01587-9ASS1-derived argininosuccinate promotes purine nucleoside synthesis in B cell acute lymphoblastic leukaemia
Gen Li, Zhijun He, Mingjie Liu, Junhua Zhao et al.
2026-09-07 · DOI: 10.1038/s42255-026-01586-wAuthor Correction: Combined alcohol and cannabinoid exposure leads to synergistic toxicity by affecting cerebellar Purkinje cells
Guichang Zou, Jing Xia, Heyi Luo, Dan Xiao et al.
2026-09-04 · DOI: 10.1038/s42255-026-01602-zGut microbiota-derived imidazole propionate promotes primary sclerosing cholangitis via p38 signalling
Antonio Molinaro, Peder Rustøen Braadland, Guido Carpino, Alba Carreras et al.
2026-09-04 · DOI: 10.1038/s42255-026-01600-1Author Correction: Impaired cAMP–PKA–CREB1 signalling drives mitochondrial dysfunction in skeletal muscle during cancer cachexia
Elia Angelino, Lorenza Bodo, Roberta Sartori, Valeria Malacarne et al.
2026-09-04 · DOI: 10.1038/s42255-026-01622-9UBE2N flips the switch on for mitophagy and off for MASH
Emily D. Madigan, Sandro L. Matos, Michael J. Jurczak
2026-09-04 · DOI: 10.1038/s42255-026-01604-xUBE2N deficiency contributes to MASH development via p62-regulated mitophagy and PANoptosis
Feng Wang, Jin Lee, Jeong-Su Park, Meizhou Huang et al.
2026-09-04 · DOI: 10.1038/s42255-026-01590-0Author Correction: Targeting MondoA–TXNIP restores antitumour immunity in lactic-acid-induced immunosuppressive microenvironment
Nannan Xu, Yemin Zhu, Yichao Han, Qi Liu et al.
2026-09-02 · DOI: 10.1038/s42255-026-01608-7Publisher Correction: Cell-specific DNA methylation in human alpha and beta cells regulates gene expression in type 2 diabetes
Jones K. Ofori, Sabrina Ruhrmann, Axel Lindström, Alexander Perfilyev et al.
2026-09-02 · DOI: 10.1038/s42255-026-01610-zA brief guide to the identification and characterization of secreted molecules
Avinash D. Londhe, Niels B. Danneskiold-Samsøe, Katrin J. Svensson
2026-08-31 · DOI: 10.1038/s42255-026-01607-8Neutrophil proteases put the brakes on visceral beiging
Marcus J. Tol, Rinke Stienstra
2026-08-31 · DOI: 10.1038/s42255-026-01606-9Neutrophil serine proteases inhibit thermogenic capacity of visceral white adipose tissue
Lufengzi Yuan, Xiaoping Wu, Jiuyu Zong, Mengxue Jiang et al.
2026-08-31 · DOI: 10.1038/s42255-026-01598-6Multi-organ proteomic atlas of obesity regression in male mice
Felix Boel, Ellen Gammelmark Klinggaard, Vyacheslav Akimov, Daniel Hansen et al.
2026-08-21 · DOI: 10.1038/s42255-026-01599-5A proteomic atlas of obesity and weight loss
Natasha D. Suriani, Matthew J. Watt
2026-08-21 · DOI: 10.1038/s42255-026-01601-0The hallmarks of skeletal muscle health
Anna Vainshtein, Bert Blaauw, Katrien De Bock, Pura Munoz-Canoves et al.
2026-08-20 · DOI: 10.1038/s42255-026-01595-9Isoleucine and valine promote prostate cancer progression via propionyl-CoA-mediated cholesterol metabolism
Zhongchi Li, Shuchen Liu, Wenbing Jin, Leyi Xiao et al.
2026-08-20 · DOI: 10.1038/s42255-026-01583-zIntestinal cGAS–STING–IFN signalling promotes obesity by downregulating microbiota-derived IAA in male mice
Jiangming Deng, Wen Meng, Yuanqin Yang, Ting Xiao et al.
2026-08-19 · DOI: 10.1038/s42255-026-01562-4Author Correction: Maternal exercise prevents metabolic disorders in offspring mice through SERPINA3C
Yang Li, Ruo-Ying Li, Jie-Ying Zhu, Min Chen et al.
2026-08-16 · DOI: 10.1038/s42255-026-01609-6Cold sensing and the parabrachial nucleus
Óscar Freire-Agulleiro, Miguel López
2026-08-13 · DOI: 10.1038/s42255-026-01571-3Reviews
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Version History
April 20, 2025 at 10:39 am
April 20, 2025